Cuban Magic Mushrooms

Cuban Magic Mushrooms

Buy Cuban Magic Mushrooms, is often referred to as the “truest” cubensis in existence because this was the first discovered cubensis in the early 1900s by F.S. Earle (an American mycologist who often worked in Cuba).

The Cuban Magic Mushrooms are known to have skinny stems and small orange-brown caps. They may appear to be small but are deceptively potent. Because of its deep rooted history this strain is considered an royalty in the world of psychedelics.

After 10-30 minutes of consuming Cuban shrooms you will feel your mood enhanced with euphoria and excitement. Depending on dosage you will experience mild to intense visual enhancements. Things may seem like they are breathing, the nature around you will feel more alive and you will find yourself in introspective thought. Music and art will look and feel different and you will have a higher appreciation and you may relate the music or art to yourself on a more personal level. The most common museum dose (0.5-1.5g) and moderate dose (2-3.5g) should provide you with a 3-6 hour trip. Please read our FAQ section for more details.

More than one way to alter brain signaling

The first approach was to completely knockout all mGluR2 receptors in a rat’s brain from birth, something the researchers achieved by using a mutant rat line that has this deficit from birth. The other approach used a targeted gene editing approach that aimed to remove mGLuR2 neurons specifically from mature rats’ addiction pathways.

The two approaches produced drastically different results. While the targeted approach reduced cognitive flexibility in a similar way to extended alcohol exposure, the global knockdown, unexpectedly, didn’t affect the rats’ behavioral performance.

Meinhardt explained that the team suspected this finding may be due to the plastic mammalian brain: “This global knockout is already present in early development, and since the brain is very plastic, during development, the brain can adapt to many, many processes. So, the idea is that receptors such as the mGluR3, which is very similar to the mGluR2, might take over the job of the mGluR2 when the brain says, ‘Okay, there are no mGluR2 left, then we have to compensate for that’,” he says.